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Diabetic and corticosteroid-impaired models are used to characterize these mechanisms under conditions of disrupted tissue repair

Notably, chemicals such as chromium, cadmium, and arsenic rely on GSH metabolism for antioxidant activity, as evidenced by increased in vitro toxicities upon depletion of intracellular GSH levels, emphasizing the indispensable role of GSH in detoxification processes facilitated by GSTs

However, it still lacks long-term human clinical data on its efficacy and safety profile

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