glp-1 insulin resistance Your body was making long before any drug existed. is why it stopped working. GLP-1 is your gut's satiety hormone. After every meal, your intestinal L-cells are supposed to GLP-1s and PMOS: PCOS, Insulin

But it barely outperformed an existing drug

Well, if we can start giving you the ability to bring that back to a physiologic state like you were when you were 30, thats a pretty amazing thing if you can do that

Their main mechanism is to delay gastric emptying (taking longer for food to pass from the stomach to the small intestine), lowering blood sugar, and regulating insulin

[DOI] [PMC free article] [PubMed] [Google Scholar] 61.Puciennik K, Siciska P, Misztal W, Bukowska B

Plosker, G

MTRR activity can also be significantly impacted by heavy metals (including mercury) and nitrous oxide (in anaesthetics and laughing gas), reducing availability of methyl B12