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Key mechanisms: Directly inhibits NF-B the central transcription factor governing the body's inflammatory cascade Suppresses MAP kinase signalling pathways Reduces pro-inflammatory cytokines including TNF-, IL-1, and IL-6 Acts via PepT1 transporters in intestinal epithelial and immune cells, targeting gut-resident inflammation at the tissue level The evidence: A landmark study published in Gastroenterology demonstrated that nanomolar concentrations of KPV inhibit NF-B and MAP kinase inflammatory pathways and reduce pro-inflammatory cytokine secretion and that oral administration of KPV reduces the incidence of chemically induced colitis in mice as measured by both histology and cytokine expression
Diabetes (2001) 50(6):134450
Biochimie 92(5):464474 Boanca G, Sand A, Barycki JJ (2006) Uncoupling the enzymatic and autoprocessing activities of Helicobacter pylori gamma-glutamyltranspeptidase
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Clinical trial outcomes do not guarantee any specific individual result
Accessed April 28, 2020
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