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ACSL4 is required to inhibit GPX4-induced ferroptosis, while ALOX12 is dispensable, whereas not required in p53-mediated ferroptosis, and therefore p53 promotes ferroptosis through a GPX4-independent pathway

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Instead, multiple complementary mechanisms are at play: some strains compete with Corynebacterium on the skin

The Source: This report includes information from The Associated Press.