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Grunberger G, Sherr J, Allende M, et al

2019 Jan 10;84(3):20924

Senescent fibroblasts in diabetic wounds exhibit ferroptosis resistance due to defective ferritinophagy, but upregulating the expression of the ferritin autophagy-related molecule NCOA4 increases their susceptibility to ferroptosis and improves wound healing [81]