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Mechanistically, BPC-157 activates VEGFR2 via the PI3K-Akt-eNOS axis to promote angiogenesis, stimulates ERK1/2 signalling to enhance fibroblast proliferation and migration, and modulates the NO system to regulate vascular tone

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BACH1 protein increased, even though BACH1 messenger RNA did not, suggesting that the change occurred mainly at the protein-stability level