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Substituting GIPRs C-terminal tail with that of GLP-1R significantly increased both the rate and extent of agonist-mediated endocytosis, suggesting a role for GIPRs C-terminal region in either its slower internalization or rapid recycling back to the plasma membrane

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Therefore, compounded tirzepatide is associated with significant potential risks

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For bone biology studies, glucocorticoid receptor antagonist controls distinguish GH-specific from residual cortisol-mediated skeletal effects