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doi: 10.1038/s41577-020-0402-6 42 ChumakovK.AvidanM

GHK further inhibited TGF-1, p-Smad2, p-Smad3, and IGF-1 expression, indicating its potential as a subject of further research in pulmonary fibrosis by targeting the TGF-1/Smad 2/3 and IGF-1 pathways

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It is a regulatory shift that appears to develop over the course of a treatment series