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Previous studies demonstrated that acylcarnitines activate pro-inflammatory signaling pathways, resulting in undesirable consequences of dysregulated FAO in mitochondria [33, 46, 68]

This review aims to synthesize emerging evidence on how GSH depletion contributes to impaired immune and antioxidant activity in neurodegenerative diseases, such as Parkinsons Disease (PD), Alzheimers Disease (AD), and multiple sclerosis (MS)

Strategies include fractionated dose, chronotherapy, and real-time monitoring [238, 239]

This is especially true for patients recovering from body contouring after major weight loss

Autophagy and ferroptosis are distinct yet intimately linked cell death modalities, with redox status playing a crucial role in their regulation (13,14)