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Thus, a role for the tissue-specific regulation of LPL activity seems to be plausible, especially when LPL activity channels fatty acids to adipose tissue for storage in the fed state and to muscle tissues as energy fuel during times of food deprivation such as fasting [129]

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Overall, the MCODE data not only show the relationships of the differential gene expression in Parkinsons and various NDs, but also specifically demonstrate enrichment of the oxidative phosphorylation pathway

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Metabolic dysfunction often accompanies muscle wasting conditions

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