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GLP-1 receptor agonists exert neuroprotective effects primarily by inhibiting microglial activation, the primary effector cells of neuroinflammation, thus reducing the production of proinflammatory cytokines (e.g., TNF- , IL-1) and alleviating inflammatory damage to brain parenchyma

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The working principle is as follows: The growth hormone causes the liver to manufacture IGF-1, which then takes charge of many of GHs anabolic effectssuch as encouraging cell growth, protein synthesis, and tissue repair

Since it influences metabolic pathways, unknown downstream effects are possible

Chin J Cancer Res

Endoplasmic reticulum stress in pancreatic -cell dysfunctionality and diabetes mellitus: a promising target for generation of functional hPSC-derived -cells in vitro