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GLP-1 receptor agonists enhance glucose-dependent insulin secretion and promote satiety, whilst SGLT2 inhibitors block renal glucose reabsorption causing urinary glucose excretion

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3.2 Oncogenes regulate Gln metabolism Some oncogenes have the ability to impact tumor development by modulating key transporters and enzymes in Gln metabolism ( Table 2 )

Signals from the gut influence appetite, satiety, and energy balance through: The vagus nerve Hormonal signaling Microbial metabolites Key metabolic hormones produced or influenced by the gut include: GLP-1 (glucagon-like peptide-1) enhances insulin secretion and promotes satiety Peptide YY (PYY) signals fullness Ghrelin stimulates hunger When gut health is disrupted, these signals can become dysregulated, potentially contributing to overeating or impaired metabolic control