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Likewise, such neuroinflammation can interact with neurotransmitters, elevate procoagulant activity and thrombosis, and cause endothelial damage, resulting in neurovascular coupling (NVC) dysfunction (363)

This manuscript investigates the pathogenesis of CRC and ferroptosis, analyses the signalling pathways involved in ferroptosis and the metabolic mechanisms implicated in ferroptosis in CRC

Before the approval of PT-141, there were limited treatment options for HSDD

Limitations: Evidence remains limited and primarily anecdotal

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Identifying these mutations enables the clinician to target patients to prescribe antioxidant protection regimes or chemotherapy regimens that will overcome these genetic defects [17]